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Findings from siRNA silencing and plasmid amplification of transferrin receptor (TFRC) suggested TFRC is crucial for TMP’s ability to mitigate ferroptosis and decrease LDH release, Fe 2+ buildup, and intracellular ROS

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HSYA reduces oxidative damage to HUVECs caused by H2O2 and inhibits ferroptosis during reperfusion, suggesting a protective role in heart tissues