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Through integrated proteomic and metabolomic analyses, we identified a “high-inflammation, high-oxidation, low-metabolism” triad in the hippocampus of CLP-induced SAE mice, providing novel insights into mechanisms underlying persistent cognitive deficits in SAE survivors
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Ideal candidates include those with treatment-resistant depression, individuals needing quick relief from severe symptoms, or those looking for a non-oral, integrative mental health option
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