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Concomitantly, YGS suppressed the expression of multiple proinflammatory cytokines, including IL-1 α , TNF-α, IL-1β, IL-17A, IL-18, IL-21, and IFN- γ , as well as chemokines such as CXCL9 and CXCL10, which are known to contribute to intestinal inflammation and barrier dysfunction
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