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The vasculature and kidney are the rich sources of NADPH oxidase-derived ROS, having important role in vascular damage and renal dysfunction under.[31] This system functions as an electron donor and catalyses the reduction of oxygen by NADPH which increases the generation of superoxide upregulation of NADPH oxidase in hypertensive patients.[31] The function of NADPH oxidase-derived superoxide is inactivation of NO in the reaction that forms peroxynitrite, leading to impaired endothelium dependent vasodilation